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Protease-activated receptors 1 and 4 do not stimulate G(i) signaling pathways in the absence of secreted ADP and cause human platelet aggregation independently of G(i) signaling.
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Mechanism of trypsin-induced endothelium-dependent vasorelaxation in the porcine coronary artery.
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Dual modulation by thrombin of the motility of rat oesophageal muscularis mucosae via two distinct protease-activated receptors (PARs): a novel role for PAR-4 as opposed to PAR-1.
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Protease-activated receptors 1 and 4 mediate activation of human platelets by thrombin.
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par-2, a gene required for blastomere asymmetry in Caenorhabditis elegans, encodes zinc-finger and ATP-binding motifs.
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Time used: 93 miliseconds.

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